User:Colin/Epilepsies
- Electroclinical syndromes
- Neonatal period
- Infancy
- Childhood
- Autosomal-dominant nocturnal frontal lobe epilepsy (ADNFLE)
- Benign epilepsy with centrotemporal spikes (BECTS)
- Childhood absence epilepsy (CAE)
- Epilepsy with myoclonic absences
- Epilepsy with myoclonic atonic (previously astatic) seizures
- Epileptic encephalopathy with continuous spike-and-wave during sleep (CSWS)
- Febrile seizures plus (FS+) (can start in infancy)
- Landau-Kleffner syndrome (LKS)
- Late onset childhood occipital epilepsy (Gastaut type)
- Lennox-Gastaut syndrome
- Panayiotopoulos syndrome
- Adolescence – Adult
- Autosomal dominant epilepsy with auditory features (ADEAF)
- Epilepsy with generalized tonic–clonic seizures alone
- Juvenile absence epilepsy (JAE)
- Juvenile myoclonic epilepsy (JME)
- Progressive myoclonus epilepsies (PME)
- Other familial temporal lobe epilepsies
- Less specific age relationship
- Familial focal epilepsy with variable foci (childhood to adult)
- Reflex epilepsies
- Distinctive constellations
- Gelastic seizures with hypothalamic hamartoma
- Hemiconvulsion–hemiplegia–epilepsy
- Mesial temporal lobe epilepsy with hippocampal sclerosis (MTLE with HS)
- Rasmussen syndrome
- Epilepsies that do not fit into any of these diagnostic categories can be distinguished first on the basis of the presence or absence of a known structural or metabolic condition (presumed cause) and then on the basis of the primary mode of seizure onset (generalized vs. focal)
- Epilepsies attributed to and organized by structural-metabolic causes
- Malformations of cortical development (hemimegalencephaly, heterotopias, etc.)
- Neurocutaneous syndromes (tuberous sclerosis complex, Sturge-Weber, etc.)
- Angioma
- Infection
- Perinatal insults
- Stroke
- Trauma
- Tumour
- Etc.
- Epilepsies of unknown cause
- Conditions with epileptic seizures that are traditionally not diagnosed as a form of epilepsy per se
- Benign neonatal seizures (BNS)
- Febrile seizures (FS)